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What does Sarnia, Sault Ste. Marie, Hamilton, St. Catharines, and Thunder Bay have in Common?
New study finds ‘strikingly high’ rates of cancer in some Ontario industrial cities: https://globalnews.ca/news/5321672/cancer-rates-sarnia/ . Acute myeloid leukemia (AML) is a heterogeneous disorder characterized by clonal expansion of myeloid progenitors (blasts) in the bone marrow and peripheral blood leading to cytopenia, infections and bleeding.1, 2 https://acsjournals.onlinelibrary.wiley.com/.../cncr.32034 . |
What are risk factors in developing AML?
-Radiation -increases with prior history of chemo or radiation (Alkylating Agents & Topo II Inhibitors) -Pesticides -Benzene -increases with age -increases with Multi-drug resistance -Smoking -Underlying Clonal myeloid diseases (https://www.patient-help.com/.../clonal-myeloid-disorders/) -Hematopoietic disorders -Congenital Conditions (https://www.who.int/.../fact.../detail/congenital-anomalies). |
Everyone should know that most cancer research is largely a fraud, and that the major cancer research organizations are derelict in their duties to the people who support them". ~Linus Paulin g Two-time Nobel Laureate 1901-1994
" . . . there is no disease whose prime cause is better known. That the prevention of cancer will come there is no doubt. But how long prevention will be avoided depends on how long the prophets of agnosticism will succeed in inhibiting the application of scientific knowledge in cancer." ~ Otto Warburg Two-time Nobel Laureate 1883-1970
Chapter 1: A Tear in the Matrix ttp://meditopia.org/chap2.htm Cancer cure known for 35 years?
" . . . there is no disease whose prime cause is better known. That the prevention of cancer will come there is no doubt. But how long prevention will be avoided depends on how long the prophets of agnosticism will succeed in inhibiting the application of scientific knowledge in cancer." ~ Otto Warburg Two-time Nobel Laureate 1883-1970
Chapter 1: A Tear in the Matrix ttp://meditopia.org/chap2.htm Cancer cure known for 35 years?
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Video: What Is This Life About? https://www.youtube.com/watch?v=ekqzD-qmw24 Transcendental Meditation does not work for everyone but a stressful situation, that's when you wake-out of your perceptual distortion of your reality. Energy-Medicine.html Energy Medicine - W.L.COLLINS Project Portfolio (weebly.com) |
Cancer during COVID-19
14:22: materials used for COVID-19
The answer is unfortunately, the human beings kept destroying the nature and the nature is getting its revenge. Please don't look for any other suspect. It infects type 2 epinephrine cells here and then causes a cytokine storm. Here, you see interleukins, a lot coming from the inflammatory mediator cells, and then it affects not only the lungs but also the liver, kidneys and blood. Cancer Highlights: • Cancer patients are more susceptible to Covid-19 pneumonia complications and death. • Cytokines and other cellular components scattered from fragmented cells increase in patients’ serums. • Serum samples of Covid-19 patients increase the motility of cancer cells. • Cancer cells express epithelial to mesenchymal transition driver genes within the 4th hour. • Cancer cells lose their intercellular junctions in 48 h. • Patients show increased lung metastasis in 6th months after having SARS-CoV-2 infection. Source: Two days Virtual International Conference on Advanced Materials JSS Science and Technology University https://www.youtube.com/watch?v=iqwzhnEwfg0 Streamed live on Jul 7, 2021 COVID-19 patients’ sera induce epithelial mesenchymal transition in cancer cells https://www.sciencedirect.com/.../pii/S2468294221001040クして編集する. Anti-parasite drug
Ivermectin can suppress cancer Ivermectin, a potential anticancer drug derived from an ... www.ncbi.nlm.nih.gov/pmc/articles/PMC7505114/ Ivermectin has powerful antitumor effects, including the inhibition of proliferation, metastasis, and angiogenic activity, in a variety of cancer ... search.nih.gov/search?utf8=%E2%9C%93&affiliate=nih&query=ivermectin%20cancer&fbclid=IwAR16OdMeR7df27S6Q9B1WLEgdNW6eLH7SiWajbKJ-t4mJeMNolLhRpoqrQU
Cheap Herbs that Are More Effective Than Drugs Get Proven Home Remedies for Depression, Heart Disease, Weight-Loss and Chronic Disease in This Eye Opening Documentary Why Use Plants in Your Healing Journey? (naturalmedicinesecrets.com) |
Thrombosis during COVID-19
Video: Venous Thrombosis, Asymptomatic vs Symptomatic COVID-19 Patients, Coronavirus Mental Health
www.youtube.com/watch?v=3UdOzNsTu1A Venous and arterial thromboembolic complications in COVID-19 patients admitted to an academic hospital in Milan, Italy C Lodigiani, G Iapichino, L Carenzo, M Cecconi… - Thrombosis research, 2020 - Elsevier
… and the high rate of positive VTE imaging tests among the few COVID-19 patients tested suggest that there is an urgent need to improve … In the Dutch study, only a minority of patients experienced arterial thrombotic events (n = 3). According to a Chinese study, the prevalence … The emerging threat of (micro) thrombosis in COVID-19 and its therapeutic implications JD McFadyen, H Stevens, K Peter - Circulation research, 2020 - Am Heart Assoc … African populations may account for some of the racial differences in thrombotic risk. In addition, … , studies evaluating Chinese and White populations have demonstrated that abnormal … -2 as a key player driving many molecular processes underpinning pathological thrombosis. www.ahajournals.org/doi/full/10.1161/CIRCRESAHA.120.317447 Purification and characterization of a fibrinolytic enzyme produced by Bacillus amyloliquefaciens DC-4 screened from douchi, a traditional Chinese soybean food Y Peng, Q Huang, R Zhang, Y Zhang - Comparative biochemistry and …, 2003 - Elsevier … have been eaten for several hundred years. Shiokara is a fermented fish product and has been eaten for more than 1000 years in Japan. Douchi is … At present, whether or not the purified subtilisin DFE induces the lysis of the thrombi in vivo and brings side effects are still under … www.sciencedirect.com/science/article/abs/pii/S1096495902001835 RECIPE for blood clots/Improve circulation. NOT MEDICAL ADVICE.https://www.youtube.com/watch?v=_UEOr1IUBD0
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Dr. Remi Buisson PhD (UCI) APOBEC3A expression in cancer & how it affects cancer development-
www.youtube.com/watch?v=SNAVbKD6mx0 Miracle in Japan www.youtube.com/watch?v=E1GF0H9V_1g More people in Asia particularly in Japan have a defense enzyme called APOBEC3A which is a defense enzyme that people have, that helps them to defend against viruses. |
P53 Role in disease Overview of signal transduction pathways involved in apoptosis.
A micrograph showing cells with abnormal p53 expression (brown) in a brain tumor. p53 immunostain. If the TP53 gene is damaged, tumor suppression is severely compromised. People who inherit only one functional copy of the TP53 gene will most likely develop tumors in early adulthood, a disorder known as Li-Fraumeni syndrome.
The TP53 gene can also be modified by mutagens (chemicals, radiation, or viruses), increasing the likelihood for uncontrolled cell division. More than 50 percent of human tumors contain a mutation or deletion of the TP53 gene.[52] Loss of p53 creates genomic instability that most often results in an aneuploidy phenotype.[53]
Increasing the amount of p53 may seem a solution for treatment of tumors or prevention of their spreading. This, however, is not a usable method of treatment, since it can cause premature aging.[54] Restoring endogenous normal p53 function holds some promise. Research has shown that this restoration can lead to regression of certain cancer cells without damaging other cells in the process.
Certain pathogens can also affect the p53 protein that the TP53 gene expresses. One such example, human papillomavirus (HPV), encodes a protein, E6, which binds to the p53 protein and inactivates it. This mechanism, in synergy with the inactivation of the cell cycle regulator pRb by the HPV protein E7, allows for repeated cell division manifested clinically as warts. Certain HPV types, in particular types 16 and 18, can also lead to progression from a benign wart to low or high-grade cervical dysplasia, which are reversible forms of precancerous lesions. Persistent infection of the cervix over the years can cause irreversible changes leading to carcinoma in situ and eventually invasive cervical cancer.
Suppression of p53 in human breast cancer cells is shown to lead to increased CXCR5 chemokine receptor gene expression and activated cell migration in response to chemokine CXCL13.[60] One study found that p53 and Myc proteins were key to the survival of Chronic Myeloid Leukaemia (CML) cells. Targeting p53 and Myc proteins with drugs gave positive results on mice with CML.[61][62]
A micrograph showing cells with abnormal p53 expression (brown) in a brain tumor. p53 immunostain. If the TP53 gene is damaged, tumor suppression is severely compromised. People who inherit only one functional copy of the TP53 gene will most likely develop tumors in early adulthood, a disorder known as Li-Fraumeni syndrome.
The TP53 gene can also be modified by mutagens (chemicals, radiation, or viruses), increasing the likelihood for uncontrolled cell division. More than 50 percent of human tumors contain a mutation or deletion of the TP53 gene.[52] Loss of p53 creates genomic instability that most often results in an aneuploidy phenotype.[53]
Increasing the amount of p53 may seem a solution for treatment of tumors or prevention of their spreading. This, however, is not a usable method of treatment, since it can cause premature aging.[54] Restoring endogenous normal p53 function holds some promise. Research has shown that this restoration can lead to regression of certain cancer cells without damaging other cells in the process.
Certain pathogens can also affect the p53 protein that the TP53 gene expresses. One such example, human papillomavirus (HPV), encodes a protein, E6, which binds to the p53 protein and inactivates it. This mechanism, in synergy with the inactivation of the cell cycle regulator pRb by the HPV protein E7, allows for repeated cell division manifested clinically as warts. Certain HPV types, in particular types 16 and 18, can also lead to progression from a benign wart to low or high-grade cervical dysplasia, which are reversible forms of precancerous lesions. Persistent infection of the cervix over the years can cause irreversible changes leading to carcinoma in situ and eventually invasive cervical cancer.
Suppression of p53 in human breast cancer cells is shown to lead to increased CXCR5 chemokine receptor gene expression and activated cell migration in response to chemokine CXCL13.[60] One study found that p53 and Myc proteins were key to the survival of Chronic Myeloid Leukaemia (CML) cells. Targeting p53 and Myc proteins with drugs gave positive results on mice with CML.[61][62]
Fundraising |
More people start fundraisers on GoFundMe than on any other platform. Learn more
Co./Org have permanent sites that exist (Dunn with Cancer).
Co./Org have permanent sites that exist (Dunn with Cancer).
Special Events are Designed to:
1. Raise needed funds; 2. Engage involved donors; 3. Encourage stewardship of existing donors; 4. Provide entry point for new donors; 5. Share mission/advocacy Annual Campaigns Assisted campaign effort that is repeated each year to underwrite annual operating budget |
Revenue
Major Gift large gift defined by dollar amount. Often considered between $10,000 and $100,000 Principle Gift large major gift, $1 million+ Leadership Annual Gift large for annual gift, $1,000+ Annual Gift relatively small gift repeated on regular/annual basis Prospect Research The use of resources to uncover signs of wealth in publicly available data. Data is then used to qualify and rate prospect. |
Defending Diction: Charity and Altruism http://themendenhall.com/2011/08/08/defending-diction-charity-and-altruism/ Exploitation
- The act of employing to the greatest possible advantage; 2. Utilization of another person or group for selfish purposes; 3. An advertising or publicity program.
The three C's?
Connection (emotional to org), Concern (recognition of important/values), Capacity (financial ability) Applying the three C's? The 3 C's determine a person's motivation or incentive to give. You need at least two and they must together to have someone give. You must identify and cultivate these in your donors. Relationships with donors are built on these. Qualities of a planful special event set definitions of success; have clear goals; mission and $ oriented; committed board and committee |
Ways of Giving:
Giving Circles Groups of donors pool their gifts and collectively decide where to direct them. Increases impact beyond 1 donor.
Giving Circles are popular amongst women, minorities, people under 40.
Giving Circles are popular amongst women, minorities, people under 40.
- Pros of giving circles are donor control, diversity, social aspect, giving more.
- Cons of giving circles are may generate smaller amount, volunteering decreases, may not contribute to larger social change.
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9 Things Boards do in fundraising
1. Give; 2. Represent the community of the org; 3. Identify prospects; 4. Cultivate prospects; 5. Introduce prospects to org; 6. Site visits with staff and prospect; 7. Make the ask; 8. Involved in saying thanks; 9. Personal notes on annual appeals
Bottom of Donor Pyramid
Bottom: largest group - annual giving cultivate through identification and informing; give through special events, memberships, direct mail, annual campaign, online and social media
1. Give; 2. Represent the community of the org; 3. Identify prospects; 4. Cultivate prospects; 5. Introduce prospects to org; 6. Site visits with staff and prospect; 7. Make the ask; 8. Involved in saying thanks; 9. Personal notes on annual appeals
Bottom of Donor Pyramid
Bottom: largest group - annual giving cultivate through identification and informing; give through special events, memberships, direct mail, annual campaign, online and social media
GENETICS and Cancer
It's Alive, But Is It Life: Synthetic Biology and the Future of Creation
https://www.youtube.com/watch?v=rU_pfCtSWF4 |
Genetically-modified babies
www.youtube.com/watch?v=b0HvLaXOhEY The researcher, He Jiankui of Shenzhen, said he altered embryos for seven couples during fertility treatments, with one pregnancy resulting thus far. He said his goal was not to cure or prevent an inherited disease, but to try to bestow a trait that few people naturally have: an ability to resist possible future infection with HIV, the AIDS virus. |
2018. Scientist claims he helped create world's first genetically-modified babies
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Big Pharma - How much power do drug companies have? | DW Documentary
www.youtube.com/watch?v=-z_W3yRA9I8&t=37s
www.youtube.com/watch?v=-z_W3yRA9I8&t=37s
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36:11
…a new gene therapy to fight cancer is now marketed by swiss company Novartis to treat leukemia its name is KYMRIAH its price is $320,000 euros, per patient. The therapy was discovered by a team of publicly funded university researchers at the University of Pennsylvania but flexing its financial muscles Novartis became co-owner of the patent professor of hematology jean-Paul Vernon is a renowned expert in blood cancers techniques. There's a whole technique involved using gene therapy a gene is introduced which allows t cells to target diseased cells and enable them to be destroyed it's a very interesting system but there's no justification for charging 350 000 euros if state structures in France went along with it it would doubtless cost 30 or 40 000 but not 350 000 euros. |
THE SUGAR JUNKIE
The Warburg effect which describes the remarkable dependence of tumors on non oxidative fermentation of sugar into lactic acid glycolysis was first described in 1926. The Warburg-Effect - Glucose Metabolism in Cancer www.youtube.com/watch?v=Kjx-pXRbnbY |
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Cancer-Associated Isocitrate Dehydrogenase Mutations
www.youtube.com/watch?v=pj0LetF3ERM Specific mutations in the isocitrate dehydrogenase gene IDH1 have been found in several brain tumors including astrocytoma, oligodendroglioma and glioblastoma multiforme. ORGANIC SOURCES OF: Chemid - ISOCITRATE DEHYDROGENASE: - Plant - Erythroxylum coca (Common name: Coca) - Plant - Glycine max (Common name: Soybean) - Latex Exudate - Papaver somniferum (Common name: Poppyseed Poppy; Opium Poppy) - Sprout Seedling - Triticum aestivum (Common name: Wheat) Chemid - NADP-ACTIVE-ISOCITRATE-DEHYDROGENASE: - Plant - Glycine max (Common name: Soybean) Chemid - Succinate-Dehydrogenase-Inhibitor: - Plant - Allicin (Common name: Garlic) |
Dr. Eric Berg
In this video, we’re going to talk about plants and cancer cells. Some plants have the ability to cause cancer cells to commit suicide. www.facebook.com/drericberg/ |
How to support the bodies defense system at a cellular level
Glutathione
Environmental toxins are absolutely crucial to avoid if you want to keep your glutathione stores at a good level. 04:38 Then also chronic exercise, people who are exercising an enormous amount, far beyond your bodies compacity. You are going to deplete glutathione levels. |
Nutrients such as milk thistle, selenium and alpha lipoic acid ...
www.youtube.com/watch?v=0R8jixulCL8 |
Low Glutathione
Many times, when people are sick and they have a hard time burning fat off their body, losing weight, and weight loss resistance. One of the things they typically do is they will go to the gym and work their heart out, but essentially what they see is they get sicker. 05:05 So, the harder they work in the gym the more fatigue they have, less energy, the more inflammation, and the sicker they become. |
Correlation Exercise Increase in Nuclear Reactor Accidents (NRAs), increase cancer rate
Between Nov. 1955 - July 2018 there were
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Correlation:
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The problem:
(A) If produced nuclear wastes are not properly transported to safe sites for strict management, the natural environment will be destroyed from pollution. Since the public is concerned with safe transport of nuclear wastes [1], [2], [3], [4], [5], [6], the problem of choosing an appropriate route for nuclear waste transport should be handled with caution.
(B) “Climate change” A potential risk factor for cancer ? Heavy Metals - Mercury
(C) Due to the disposal of high-activity nuclear wastes in the oceans [1], ingestion of fish is identified as the principal pathway to excess cancer risk.
(D) Increase incidents of young children developing Papillary carcinoma.
(E) [1], [2], [3], [4], [5], [6],
(B) “Climate change” A potential risk factor for cancer ? Heavy Metals - Mercury
(C) Due to the disposal of high-activity nuclear wastes in the oceans [1], ingestion of fish is identified as the principal pathway to excess cancer risk.
(D) Increase incidents of young children developing Papillary carcinoma.
(E) [1], [2], [3], [4], [5], [6],
WORLD CANCER REPORT Cancer research has improved the lives of millions of people throughout the world, but the results are uneven and different Cancers affect people in different ways depending upon where they live. Below are the World Rankings for selected Cancers that are the most common. See USA Cancer Report here: https://www.worldlifeexpectancy.com/usa-cancer-report Link: List of nuclear power accidents by country |
61 Nuclear reactor accidents (NRAs) in the USA/US
FOUR NRAs
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Three NRAs
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TWO NRAs
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One NRAs
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The value of r measures the strength of a correlation based on a formula, eliminating any subjectivity in the process.
26 United States Nuclear Plants https://consthagyg.blogspot.com/2020/01/26-nuclear-plants-us-map.html |
The Art of Disease
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Global and regional probabilities of major nuclear reactor accidents
We have found that even when individual reactor safety is assumed to be very high, the chance of another major accident of INES level 6 or 7 remains sizeable in all regions of the world. Improvements in individual reactor safety can be expected to reduce overall accident probability considerably, but such improvements will likely become ever more costly. It is certainly possible to reduce humanity’s carbon emissions by energy policies that rely on nuclear power, but the relatively high probability of another Chernobyl or Fukushima anywhere on the globe and the unresolved question of nuclear waste management suggest that the burden on future generations will merely be shifted from atmospheric CO2 to high-level radioactive waste and long-term nuclear contamination of accident sites, their surroundings, and potentially way beyond. In light of this, renewable energies, in particular wind (Bandoc et al., 2018) and solar energy (Prăvălie et al., 2018) seem to be the better energy policy options to cover humanity’s energy supply sustainably.
Source:
Global and regional probabilities of major nuclear reactor accidents
John-OliverEngler. Journal of Environmental Management, Volume 269, 1 September 2020
Source:
Global and regional probabilities of major nuclear reactor accidents
John-OliverEngler. Journal of Environmental Management, Volume 269, 1 September 2020
Nuclear Fuel in a Reactor Accident
Release of volatile and gaseous fission products during a core-melt event is effectively instantaneous, and this contributes dramatically to the near-term environmental impact due to their atmospheric dispersion. Water that interacts with damaged fuel will transport radionuclides that present both short-term and longer-term environmental risk, beginning with the core-melt event and potentially continuing for many years if the damaged fuel is not adequately isolated from the environment. The events at Fukushima Daiichi have demonstrated the importance of water-borne radionuclide transport after a core-melt event, with as much as 27,000 terabecquerels of 137Cs being released to the ocean (8). The Future Design of fuel assemblies and reactors that are better able to withstand potential accidents.
Source:
Nuclear Fuel in a Reactor Accident
Peter C. Burns et al.
Science 335, 1184 (2012)
https://web.mit.edu/mission/www/m2018/pdfs/japan/reactor_accident.pdf
Source:
Nuclear Fuel in a Reactor Accident
Peter C. Burns et al.
Science 335, 1184 (2012)
https://web.mit.edu/mission/www/m2018/pdfs/japan/reactor_accident.pdf
Cancer arises when the DNA changes via: |
What do they do with the 18 month old Fuel Cells?
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1. some changes are hereditary
4. certain viral infections, opportunistic infections
The evolution of resistance and tolerance as cancer defences |
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[HTML] Antihypertensive Drugs and the Risk of Cancer: A Nationwide Cohort Study
Cumulative Radiation Exposure and Cancer Risk Estimates in Emergency Department Patients Undergoing Repeat or Multiple CT
A small proportion (1.9%) of emergency department patients undergoing CT of the neck, chest, abdomen, or pelvis have high cumulative rates of multiple or repeat imaging. Collectively, this patient subgroup may have a heightened risk of developing cancer from cumulative CT radiation exposure.
Read More: https://www.ajronline.org/doi/full/10.2214/AJR.08.1351
A small proportion (1.9%) of emergency department patients undergoing CT of the neck, chest, abdomen, or pelvis have high cumulative rates of multiple or repeat imaging. Collectively, this patient subgroup may have a heightened risk of developing cancer from cumulative CT radiation exposure.
Read More: https://www.ajronline.org/doi/full/10.2214/AJR.08.1351
Barrett's Esophagus - Esophageal Cancer
Here's What We Know
Barrett's esophagus is a condition in which the flat pink lining of the swallowing tube that connects the mouth to the stomach (esophagus) becomes damaged by acid reflux, which causes the lining to thicken and become red. Symptoms: Heartburn Having Barrett's esophagus may raise your risk of getting esophageal cancer. There is no cure for Barrett's esophagus. Your care plan will try to stop any more damage by keeping acid reflux out of your esophagus. |
What Many Don't Know 14.2.1.2 Arachidonic Acid Pathway Arachidonic acid pathway is a central regulator of inflammatory response. Gastric acid and duodenal bile acids may contribute to carcinogenesis in BE through activation of this pathway. Low pH and bile acids induce cyclooxygenase-2 (COX-2), which is a central enzyme of the arachidonic acid pathway both in the human BE ex vivo culture model and the EAC cell lines [32,33]. Expression of COX-2 increases concomitantly with neoplastic progression in BE and this increase supports an association between the arachidonic acid pathway and the development of EAC [32–35]. The COX-2 enzyme catalyzes conversion of arachidonic acid to different prostaglandins such as prostaglandin E2 (PGE2). PGE2 induces proliferation of Barret’s epithelial cells and its inhibition may slow their growth [36]. PGE2-induced cell proliferation leads to accumulation of replicative errors in premalignant Barrett’s cells. Furthermore, it inhibits tumor surveillance through natural killer cell activity of PGE2. Chronic induction of PGE2 might facilitate accumulation of abnormal cells that have genomic instability and thus inhibition of components of arachidonic acid pathway probably will inhibit carcinogenesis [37]. Chemoprevention of Barrett’s Esophagus and Adenocarcinoma Ebubekir Şenateş, in Barrett's Esophagus, 2016 https://www.sciencedirect.com/science/article/pii/B9780128025116000144````````````` Video: Arachidonic Acid Pathway...Best Explanation! https://www.youtube.com/watch?v=gnz841lGa7Q |
Cancer Research Institute
https://www.cancerresearch.org/blog/september-2019/blood-cancer-awareness-month-2019-immunotherapy |
Chemotherapy, Surgery and Radiation can be Obsolete Treatments for Cancer
In purple, patient survival data from 8 different types of cancer and the bars represent survival time taken from the era in which there was only chemotherapy, surgery, or radiation available in but starting in 2004 when antiangiogenic therapies first became available (in yellow).
What is the advantages of antiangiogenic therapy?
* Less drug resistance. * Effective against a broad range of cancers. * Work well below the maximum tolerated dose--> longer treatments. * Supplement to chemo- or radio-therapy * Selectively target dividing blood vessels fewer side effects (relatively nontoxic). |
What is a disadvantage of antiangiogenic therapy?
* Don't directly attack the tumor cell--> tumor are not killed. Therefore, this type of therapy may need to be administered over a long period so Start Today. |
Royal Raymond Rife Suppressed Medical Technology
www.youtube.com/watch?v=2fh0RJczTAc Cumulative Radiation Exposure and Cancer Risk Estimates in Emergency Department Patients Undergoing Repeat or Multiple CTRichard T. Griffey1 and Aaron Sodickson2 CONCLUSION. A small proportion (1.9%) of emergency department patients undergoing CT of the neck, chest, abdomen, or pelvis have high cumulative rates of multiple or repeat imaging. Collectively, this patient subgroup may have a heightened risk of developing cancer from cumulative CT radiation exposure. Read More: https://www.ajronline.org/doi/full/10.2214/AJR.08.1351 |
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Triple-negative Breast Cancer | Details, Diagnosis, and Signs
Julia Levy(born 1934) Canadian Pathologist. Pioneer in the field of photodynamic therapy. en.wikipedia.org/wiki/Julia_Levy
She has perfected a chemotherapeutic drug that, unlike most chemotherapy drugs, is activated only when it is exposed to light. After administering the light-sensitive biochemical agent, a thin fiber-optic cable is threaded into the patient's body and directed to the exact spot on the lung, liver, pancreas, or other organ where the tumor is growing. Laser light is then beamed through the fiber-optic cable to the cancer site. When the laser hits the spot of the organ or tissue where the cancer is to be found, it activates the light-sensitive biochemical drug, which then attacks the cancer at only that one place. In this way, the treatment does not weaken the patient by attacking healthy cells like other broad-based chemical therapy treatments. Levy has extended her light-sensitive biochemical approach to treatments of arthritis, AIDS, and age-related vision loss. www.youtube.com/watch?v=lTIHY5iIF0Q
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In Sight by Julia Levy | Book Trailer | Rotman-UTP Publishing
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Genes make up less than 2% of human DNA; the remaining DNA (mostly Bacterial DNA) has important but still largely unknown functions.
View or Download The Human Genome Landmarks Poster Of the 2%, Genetic Birth Defect (GBD) may account for less than 5%, if you weren't born with GBD than you can not owe it to genetics soley; and behavior may have contributed to it but again it is not the root cause. https://www.ted.com/talks/margaret_heffernan_dare_to_disagree#t-7777 |
In 1953 the Medical Research Council allocated funds to her pioneering study of x-rays as a cause of childhood cancer, which she worked on from 1953 until 1956. Her results were initially regarded as unsound. Her findings on fetal damage caused by x-rays of pregnant women were eventually accepted worldwide and the use of medical x-rays during pregnancy and early childhood was curtailed as a result (although it took around two and a half decades).[2] Stewart retired in 1974.
https://en.wikipedia.org/wiki/Alice_Stewart |
These 8 potent nutrients KILL cancer cells with no side effects
Wednesday, June 27, 2018 by: Vicki Batts
Tags: anticancer foods, cancer, cancer prevention, cruciferous vegetables, curcumin, food fights cancer, ginger, good food, goodhealth, healthy eating, medicinal foods, natural remedies, nutrition, phytochemcials, prevention, quercetin, supplements, vitamin C, vitamin D
(Natural News) Can you prevent cancer with diet and lifestyle changes?
For the overwhelming majority of people, the answer is an emphatic, “Yes.” Despite what the cancer industry would like you to believe, food is medicine. And eating the right foods can help ward off cancer. Studies have shown that upwards of 90 percent of all cancers can be traced back to dietary, lifestyle and environmental factors — which means very few cancers are just a case of bad luck and genetic lottery.
The links between lifestyle, diet, and cancer have been well-established. A 2008 study led by scientists at the University of Texas in Houston found that “cancer is a preventable disease that requires major lifestyle changes. More recent studies have continued to hit on these correlations, such as this 2016 study from researchers at Harvard University, which concluded, “A substantial cancer burden may be prevented through lifestyle modification.
While you can’t go wrong with a plant-based diet, there are some fruits, vegetables and herbs that are highly regarded for their exceptional anti-cancer effects. These amazing benefits are derived from a number of plant nutrients:
1. Gingerols
Gingerols are found in the popular ginger root — and they pack quite the anti-cancer punch. There are a number of gingerols and other cancer-fighting compounds found in ginger root, such as zingerone, paradols and 6-shogaol. 6-gingerol, in particular, is highly regarded for its anti-cancer effects.
A 2016 study from the Centre for Plant Biotechnology and Molecular Biology in India declared 6-gingerol as the most potent anti-cancer compound found in the ginger root. Other studies have shown compounds in ginger are able to suppress the growth and spread of cancer cells in breast and colorectal cancers.
2. Sulforaphane
Sulforaphane is a compound found in cruciferous vegetables like broccoli, Brussels sprouts, kale, mustard greens and cabbage. An organosulphate compund, sulforaphane has been extensively studied for its host of health benefits. As Natural Health 365 reports:
This amazing phytochemical destroys cancer cells, reduces inflammation that contributes to cancer, helps to prevent DNA changes that can lead to cancer, and even helps neutralize an enzyme that coverts procarcinogens into active carcinogens – pretty impressive benefits to be gained from a daily helping of humble Brussels sprouts.
3. Vitamin C
Vitamin C is known for many things, like immune system support and wound healing. But did you know this essential nutrient also fights cancer? Studies show that vitamin C can reduce inflammation, fight free radicals and, in the right dose, it can even kill off cancer cells.
4. Quercetin
Quercetin is found in abundance in apples, grapes, red onions, and tomatoes. Studies show that a diet high in this plant nutrient can reduce cancer risk by up to 50 percent. This antioxidant nutrient can help protect cells and DNA from the damage caused by free radicals. Studies have shown that quercetin stops cancer in its tracks, killing off cancer cells before they have the chance to spread.
5. Vitamin D
Vitamin D is an essential nutrient known for its benefits for bone health and depression, but it helps prevent cancer, too. Studies have shown that it reduces cancer risk across the board, but it is especially beneficial for warding off prostate, colorectal and liver cancers.
6. Curcumin
Curcumin is the active compound found in turmeric. Study after study has shown that this powerhouse of a nutrient can fight cancer at almost every turn. Earlier this year, it was revealed that someone cured their stage-3 myeloma (bone cancer) with turmeric and nothing else — a feat so great it was published in the British Medical Journal. Even proponents of the corrupt cancer industry couldn’t ignore that.
7. Proanthocyanidins
Found in grape seed extract, proanthocyanidins are known to cut the risk of cancer by a substantial margin — and more importantly, these potent nutrients are capable of slowing cancer growth and killing off cancer cells. Proanthocyanidins have also been shown to help prevent liver damage caused by chemotherapy.
8. Oleocanthal
We all know olive oil is good for us — but just how good is it? Oleocanthal is a plant nutrient found in olive oil that has the power to rupture cancer cell walls, leaving them to be destroyed by their own enzymes. https://www.naturalnews.com/2018-06-27-these-9-potent-nutrients-kill-cancer-cells-with-no-side-effects.html
Wednesday, June 27, 2018 by: Vicki Batts
Tags: anticancer foods, cancer, cancer prevention, cruciferous vegetables, curcumin, food fights cancer, ginger, good food, goodhealth, healthy eating, medicinal foods, natural remedies, nutrition, phytochemcials, prevention, quercetin, supplements, vitamin C, vitamin D
(Natural News) Can you prevent cancer with diet and lifestyle changes?
For the overwhelming majority of people, the answer is an emphatic, “Yes.” Despite what the cancer industry would like you to believe, food is medicine. And eating the right foods can help ward off cancer. Studies have shown that upwards of 90 percent of all cancers can be traced back to dietary, lifestyle and environmental factors — which means very few cancers are just a case of bad luck and genetic lottery.
The links between lifestyle, diet, and cancer have been well-established. A 2008 study led by scientists at the University of Texas in Houston found that “cancer is a preventable disease that requires major lifestyle changes. More recent studies have continued to hit on these correlations, such as this 2016 study from researchers at Harvard University, which concluded, “A substantial cancer burden may be prevented through lifestyle modification.
While you can’t go wrong with a plant-based diet, there are some fruits, vegetables and herbs that are highly regarded for their exceptional anti-cancer effects. These amazing benefits are derived from a number of plant nutrients:
1. Gingerols
Gingerols are found in the popular ginger root — and they pack quite the anti-cancer punch. There are a number of gingerols and other cancer-fighting compounds found in ginger root, such as zingerone, paradols and 6-shogaol. 6-gingerol, in particular, is highly regarded for its anti-cancer effects.
A 2016 study from the Centre for Plant Biotechnology and Molecular Biology in India declared 6-gingerol as the most potent anti-cancer compound found in the ginger root. Other studies have shown compounds in ginger are able to suppress the growth and spread of cancer cells in breast and colorectal cancers.
2. Sulforaphane
Sulforaphane is a compound found in cruciferous vegetables like broccoli, Brussels sprouts, kale, mustard greens and cabbage. An organosulphate compund, sulforaphane has been extensively studied for its host of health benefits. As Natural Health 365 reports:
This amazing phytochemical destroys cancer cells, reduces inflammation that contributes to cancer, helps to prevent DNA changes that can lead to cancer, and even helps neutralize an enzyme that coverts procarcinogens into active carcinogens – pretty impressive benefits to be gained from a daily helping of humble Brussels sprouts.
3. Vitamin C
Vitamin C is known for many things, like immune system support and wound healing. But did you know this essential nutrient also fights cancer? Studies show that vitamin C can reduce inflammation, fight free radicals and, in the right dose, it can even kill off cancer cells.
4. Quercetin
Quercetin is found in abundance in apples, grapes, red onions, and tomatoes. Studies show that a diet high in this plant nutrient can reduce cancer risk by up to 50 percent. This antioxidant nutrient can help protect cells and DNA from the damage caused by free radicals. Studies have shown that quercetin stops cancer in its tracks, killing off cancer cells before they have the chance to spread.
5. Vitamin D
Vitamin D is an essential nutrient known for its benefits for bone health and depression, but it helps prevent cancer, too. Studies have shown that it reduces cancer risk across the board, but it is especially beneficial for warding off prostate, colorectal and liver cancers.
6. Curcumin
Curcumin is the active compound found in turmeric. Study after study has shown that this powerhouse of a nutrient can fight cancer at almost every turn. Earlier this year, it was revealed that someone cured their stage-3 myeloma (bone cancer) with turmeric and nothing else — a feat so great it was published in the British Medical Journal. Even proponents of the corrupt cancer industry couldn’t ignore that.
7. Proanthocyanidins
Found in grape seed extract, proanthocyanidins are known to cut the risk of cancer by a substantial margin — and more importantly, these potent nutrients are capable of slowing cancer growth and killing off cancer cells. Proanthocyanidins have also been shown to help prevent liver damage caused by chemotherapy.
8. Oleocanthal
We all know olive oil is good for us — but just how good is it? Oleocanthal is a plant nutrient found in olive oil that has the power to rupture cancer cell walls, leaving them to be destroyed by their own enzymes. https://www.naturalnews.com/2018-06-27-these-9-potent-nutrients-kill-cancer-cells-with-no-side-effects.html
The Danger of Willful Blindness
www.ted.com/talks/margaret_heffernan_the_dangers_of_willful_blindness#t-341140
Vermiculite Gayla Benefield was just doing her job, reading meters, She uncovered an awful secret about her hometown that meant its mortality rate was 80 times higher than anywhere else in the US. But when she tried to tell people about it, she learned an even more shocking truth: People didn't want to know. In a talk that's part history lesson, part call-to-action, Margaret Heffernan demonstrates the danger of willful blindness, and praises ordinary people like Benefield who are willing to speak up. |