'Portrait of Joseph Roulin' ()
Boston, Museum of Fine Arts In the winter of 1888, Vincent van Gogh painted the portrait of his good friend and neighbor, Joseph Roulin who was born on 4 April 1841 in Lambesc located in southern France; he was a 47 year old Postman.
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Vincent Willem van Gogh
Netherlands 1853 – 1890 France (d. 37) Dutch Post-Impressionist artist Vincent's enthusiasm and gift for drawing was evident at a young age. His ability to perceive sharply and with originality was evident right from his earliest letters which contributed greatly to his autobiography. It was suggested that Vincent had a future in the trade of art dealer, a profession already practiced successfully by different members of his family but Vincent preferred to paint. He had a keen awareness of colour, particularly to blue. He had a preference for parks, flower beds, and views from a window. His brother Theo became his only trusted dealer as Vincent was not eager to release any of his works.
Before the age of 27 there were periods were Vincent chose not to draw as this time was filled with activities as preacher or spent in the depths of his great personal sorrows or roaming the forests. In 1888-1892 his work was exhibition before the public eye. The paintings were hung according to color, not chronologically. |
'Worn Out: At Eternity's Gate' (1890)
Though he believed Van Gogh was suffering from a form of epilepsy caused by too much coffee and alcohol and too little food, his doctor, Dr. Rey never made an official diagnosis.
"Lust for Life", video captures Vincent's depression. |
Ketamine for major depression: New tool, new questions
www.health.harvard.edu/blog/ketamine-for-major-depression-new-tool-new-questions-2019052216673
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Once reserved for the party scene psychedelic drugs are making a comeback in an unlikely way as a promising treatment for depression and other mental illnesses. Magic mushrooms have been legal for some palliative patients in Canada for the past year. W5 investigation reveals another
psychedelic is also quietly being supported by the government for some Veterans struggling with depression and PTSD. W5: Harnessing the power of psychedelic drug therapy https://www.youtube.com/watch?v=sB9IhQXtBLg |
Improve Your Health Through Breathing
The autorhythmicity inherent in cardiac cells keeps the heart beating at a regular pace; however, the heart is regulated by and responds to outside influences as well. Extreme stress from such life events as the death of a loved one, an emotional break up, loss of income, or foreclosure of a home may lead to a condition commonly referred to as broken heart syndrome. This condition may also be called Takotsubo cardiomyopathy, transient apical ballooning syndrome, apical ballooning cardiomyopathy, stress-induced cardiomyopathy, Gebrochenes-Herz syndrome, and stress cardiomyopathy. (1). Because stress cardiomyopathy can often mimic a heart attack, many patients may initially be treated using heart attack treatments. The treatment for stress cardiomyopathy involves largely supportive care while the heart gets stronger and the blood pressure improves. |
Once it has been identified that stress cardiomyopathy is the cause of the patient’s condition, treatments may include:
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As patients recover, they are encouraged to eat a proper diet and explore methods to manage stress, such as through exercise, supportive relationships, and meditation. (2) |
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Introduction
Mindful breathing (pranayama) is the most basic mindfulness meditation. Pranayama, or yogic breathing, is a practice in yoga that focuses on controlling the breath through breathing exercises and techniques. Several studies show that practicing pranayama help ease stress and lower levels of the stress hormone cortisol; (3,4,5,6) can lead to a decrease in symptoms of anxiety; (7,8) reduce inflammatory markers in the body and help prevent pro-inflammatory diseases; (9,10,11) decrease risk factors for heart disease; (12,13,14) decrease symptoms of depression by influencing the production of stress hormones in the body; (15,16.17) and enhance sleep quality because of its effects on melatonin and its impact on several common contributors to sleep problems. (18) |
Three Techniques are found in the following videos:
. 1. Ujjayi Pranayama (duration is 10 minutes) Video: https://www.youtube.com/watch?v=Vo_gmNHODGA 2. Bhastrika Pranayama (duration is 10 minutes) Video: https://www.youtube.com/watch?v=n2Eunbhe3NA 3. Viloma Pranayama (duration is 10 minutes) Video: https://www.youtube.com/watch?v=RUFzLVf5wL4 Count the number of breaths you take in one minute before and after (wait for 15 seconds) the exercise. Do one exercise, one day only, in the morning to see which technique works best for you. . |
Depression, the secret we share | Andrew Solomon
The chemical cure and the psychological cure both have a role to play in something that is intertwined so deep, that there is no separating it from our character and personality.
Grief is explicitly reactive. If you have a loss and you feel incredibly unhappy, and then, six months later, you are still deeply sad, but you're functioning a little better, it's probably grief, and it will probably ultimately resolve itself in some measure. If you experience a catastrophic loss, and you feel terrible, and six months later you can barely function at all, then it's probably a depression that was triggered by the catastrophic circumstances. In his video, Andrew asks, What are the mechanisms that allow people to survive and what causes some people to be more resilient than other people? |
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Benzos.
76:28: for people who take these drugs: Valium, Xanax, Klonopin, Ativan.....These drugs are particularly poisonous, toxic, addictive and suffering inducing. And if you think that you're gonna wave a magic wand with these drugs and fix your biopsychosocial problem of anxiety or whatever it is, you are being fed a bill of goods and the suffering these drugs cause is at least as bad as opioids. Maybe worse. https://www.youtube.com/watch?v=UbIhOZg_9mw Benzodiazepines, sometimes called "benzos", are a class of psychoactive drugs whose core chemical structure is the fusion of a benzene ring and a diazepine ring. As depressants—drugs which lower brain activity—they are prescribed to treat conditions such as anxiety, insomnia, and seizures. .Wikipedia |
Omicron, Depression, Imposter Syndrome, Benzos, Insomnia (w/Dr. Rachel Zoffness) (zdoggmd.com)
https://zdoggmd.com/pain-points-2/?fbclid=IwAR3LxMQgmRfNNA14T_LvHGckcxxzrsMHizXlTPSGSpcbOcqWFHvh3SXDPMk |
The cytokine hypothesis of depression:
inflammation, oxidative & nitrosative stress (IO&NS) and leaky gut as new targets for adjunctive treatments in depression.
M Maes - Neuroendocrinol Lett, 2008 - nel.edu
M Maes - Neuroendocrinol Lett, 2008 - nel.edu
There is now evidence, that inflammatory pathways play an important role in the pathophysiology of depression [3]. Multiple inflammatory biomarkers have been detected in depression:
a) increased plasma levels or production of pro-inflammatory cytokines, e.g. “monocytic” cytokines, such as interleukin-1β(IL-1β), IL-6, and tumor necrosis factor-α (TNFα), and Th-1-like cytokines, such as interferon-γ (IFNγ); and b) signs of an acute phase (AP) response, such as increased levels of AP proteins and lower serum zinc [3]. c) indicators that lowered plasma tryptophan, an established inflammatory marker, bears a strong relationship with the inflammatory biomarkers of depression [3] |
Further evidence that inflammation plays a role in depression comes from animal and human models,
a) In animal models of depression, e.g. the chronic mild stress (CMS) model, multiple inflammatory markers are detected [3,8]. b) Novel animal models of depression have been developed based on induced inflammation, e.g. administration of lipopolysaccharide (LPS) [9] and sustained administration of IL-6 [10]. c) Systemic inflammation is known to cause a central neuroinflammation with activation of brain microglia and chronically elevated pro-inflammatory reactions [11]. d) Peripheral and central administration of cytokines, such as IL-1β and IL-6, to animals may induce sickness behavioral symptoms, such as anorexia, weight loss, soporific effects, disturbances of locomotor activity and exploration, or anhedonia [12]. Interestingly, the latter symptoms are similar to the vegetative symptoms of depression, while in depressed patients the occurrence of those symptoms is significantly related to inflammatory biomarkers [3,13]. This suggests that the vegetative symptoms in depression are induced by inflammation [3,13]. e) In humans, cytokine-based immunotherapy for hepatitis-C was reported to be accompanied by depressive symptoms and full blown depression in a considerable number of patients [7]. |
Thus, the cytokine theory of depression hypothesizes that
a) depression results from an increased production of pro-inflammatory cytokines, which may be triggered by external or internal stressors; b) inflammation may induce depressive symptoms through different pathways, such as central neuroinflammation, tryptophan degradation and an increased synthesis of neurotoxic TRYCATs; 290 Copyright © 2008 Neuroendocrinology Letters ISSN 0172–780X • www.nel.edu Michael Maes c) increased O&NS may disrupt lipid membrane components and may modify protein structures thereby mounting an autoimmune response and interfere with functional proteins; d) the clinical efficacy of antidepressants is at least in part related to their anti-inflammatory activity, for example, through their interactions with the inflammatory - serotonin pathway; and d) antiinflammatory compounds including natural antiO&NS substances (NAIOSs) may augment the efficacy of antidepressants or may have antidepressive efficacy |
How Bacteria Face Depression, Recession, and Derepression AL Koch - Perspectives in biology and medicine, 1976 - muse.jhu.edu https://muse.jhu.edu/article/405611/summary
These alliances can be broadly classified according to (i) their means of transmission from generation to generation (acquisition from the environment vs. transfer via gametes from the female parent, as in bacteriocyte-insect symbioses); (ii) the physical relationship between symbiont and host (intracellular vs. extracellular); and (iii) whether the association is binary (one host and one microbial species), as is typical in invertebrates, or consortial, as in the cow rumen and human gut (2)
The Nature of Ayahuasca (2019) Documentary04:12
After working with Ayahuasca find that they do not go back to those feelings and they experience a permanent relief Same is true for anxiety issues phobias of various types irrational fears of things stress and trauma disorders post traumatic stress disorder and things of this nature |
Gen Z: The Most Anxious Generation.
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Skill ThreeSed ut perspiciatis unde omnis iste natus error sit voluptatem accusant doloremque laudantium, totam rem.
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