What is the causative agent of Tetanus? Clostridium tetani, it is an obligate anaerobe
What are the symptoms of tetanus? muscle spasms capable of breaking bone, lockjaw, and death do to suffocation
What are the signs of neonatal tetanus? for a baby, having a forced grin or raised eyebrows (35:46 into video)
What is the exotoxin that causes the symptoms of tetanus? tetanospasmin - Source: for title page image
How does tetanospasmin affect neurons? It blocks inhibitory neurons which makes muscles in capable of relaxing
What type of toxin is tetanospasmin? it is an A/B toxin
How does tetanospasmin travel to the body? it enters through deep puncture wound then the disease germinates and releases exotoxins into the bloodstream which they can carry the toxin and interactions a motor neurons then the tetanospasmin travels through retrograde transport to the spinal cord to inhibitory neurotransmitters (neurotransmitters that send signals for muscles to relax). Once the toxins bind to the nerves it's irreversible, nothing can be done about it besides your body's own healing mechanism that will allow it to grow new nerve terminals.
What is tetany? a state state of muscle contraction
How does someone contract tetanus? through a contaminated puncture wound burn or frostbite frostbite or a contaminated severed umbilical cord. It is a non-communicable disease
Who is at high risk of contracting tennis?
The elderly population who have a tendency to forget their booster shot shots, heroin users, patients with puncture wounds and lacerations, people in underdeveloped countries
What is the reservoir tetanus? soil and cows or humans can have the pathogen in their G.I. track
What is TIG? tetanus immunoglobulin
what is TIG used for? it is used to provide immediate protection to a patient after being exposed to tetanus by introducing antibodies into the bloodstream
Why is TIG used first for treatment instead of the vaccine? The vaccine takes time to develop antibodies where TIG immediately gives the body antibodies
what is DTaP? A combination vaccine which prevents diphtheria, tetanus, and pertussis
What is pertussis? whooping cough
What are the symptoms of tetanus? muscle spasms capable of breaking bone, lockjaw, and death do to suffocation
What are the signs of neonatal tetanus? for a baby, having a forced grin or raised eyebrows (35:46 into video)
What is the exotoxin that causes the symptoms of tetanus? tetanospasmin - Source: for title page image
How does tetanospasmin affect neurons? It blocks inhibitory neurons which makes muscles in capable of relaxing
What type of toxin is tetanospasmin? it is an A/B toxin
How does tetanospasmin travel to the body? it enters through deep puncture wound then the disease germinates and releases exotoxins into the bloodstream which they can carry the toxin and interactions a motor neurons then the tetanospasmin travels through retrograde transport to the spinal cord to inhibitory neurotransmitters (neurotransmitters that send signals for muscles to relax). Once the toxins bind to the nerves it's irreversible, nothing can be done about it besides your body's own healing mechanism that will allow it to grow new nerve terminals.
What is tetany? a state state of muscle contraction
How does someone contract tetanus? through a contaminated puncture wound burn or frostbite frostbite or a contaminated severed umbilical cord. It is a non-communicable disease
Who is at high risk of contracting tennis?
The elderly population who have a tendency to forget their booster shot shots, heroin users, patients with puncture wounds and lacerations, people in underdeveloped countries
What is the reservoir tetanus? soil and cows or humans can have the pathogen in their G.I. track
What is TIG? tetanus immunoglobulin
what is TIG used for? it is used to provide immediate protection to a patient after being exposed to tetanus by introducing antibodies into the bloodstream
Why is TIG used first for treatment instead of the vaccine? The vaccine takes time to develop antibodies where TIG immediately gives the body antibodies
what is DTaP? A combination vaccine which prevents diphtheria, tetanus, and pertussis
What is pertussis? whooping cough
Dr. Carrie Madej warns about coronavirus vaccines and transhumanism nanotechnology to alter your DNA
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Tetanus vaccines - 985 deaths & 23,981 injuries last 10 years
https://www.youtube.com/watch?v=grPtch4J9Ks |
You Don't Need Tetanus Vaccine after Every Injury | Know about Tetanus Vaccine in Adults | Tdap |
Manual for the Surveillance of Vaccine-Preventable Diseases https://www.cdc.gov/vaccines/pubs/surv-manual/chpt16-tetanus.html |
43:16
We can't say for sure that the vaccines are causing these things but we have now evidence that they could be and we can't say that they're not causing them and we know from toxicology that many of the chemicals that are in vaccines are stirring up the immune system in ways that are not normal and I'll get into that more in a bit. So, this is another study that I will show you from 2008. This was a Swedish study and they looked at infants after their Diphtheria, Tetanus, Acellular Pertussis, and Polio Vaccine ... |
These are babies and they got the
regular vaccines at 3 and 5 months and then they took some of their blood out and they exposed it to the whooping cough toxin which is basically what's in the vaccine and then they look to see what happened 12 years later in a genetic level (Image left) So we're definitely seeing changes on a genetic level |
. 2017 Mar;17:192-198.
doi: 10.1016/j.ebiom.2017.01.041. Epub 2017 Feb 1.The Introduction of Diphtheria-Tetanus-Pertussis and Oral Polio Vaccine Among Young Infants in an Urban African Community: A Natural Experiment Søren Wengel Mogensen 1, Andreas Andersen 2, Amabelia Rodrigues 1, Christine S Benn 3, Peter Aaby 4 pubmed.ncbi.nlm.nih.gov/28188123/ Background: We examined the introduction of diphtheria-tetanus-pertussis (DTP) and oral polio vaccine (OPV) in an urban community in Guinea-Bissau in the early 1980s. Methods: The child population had been followed with 3-monthly nutritional weighing sessions since 1978. From June 1981 DTP and OPV were offered from 3months of age at these sessions. Due to the 3-monthly intervals between sessions, the children were allocated by birthday in a 'natural experiment' to receive vaccinations early or late between 3 and 5months of age. We included children who were <6months of age when vaccinations started and children born until the end of December 1983. We compared mortality between 3 and 5months of age of DTP-vaccinated and not-yet-DTP-vaccinated children in Cox proportional hazard models. Results: Among 3-5-month-old children, having received DTP (±OPV) was associated with a mortality hazard ratio (HR) of 5.00 (95% CI 1.53-16.3) compared with not-yet-DTP-vaccinated children. Differences in background factors did not explain the effect. The negative effect was particularly strong for children who had received DTP-only and no OPV (HR=10.0 (2.61-38.6)). All-cause infant mortality after 3months of age increased after the introduction of these vaccines (HR=2.12 (1.07-4.19)). Conclusion: DTP was associated with increased mortality; OPV may modify the effect of DTP. Keywords: DTP; Diphtheria-tetanus-pertussis vaccine; Measles vaccine; Non-specific effects of vaccines; Oral polio vaccine. |
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